News

Exciting news is emerging from the latest clinical research; the innovative SuperNOVA Stent (Neurovascular Ostial Advanced Stent Retriever) has just been demonstrated to be both safe and effective for treating acute ischemic strokes in clinical trials. This is a significant development in the world of endovascular therapy, offering new possibilities for improving outcomes for survivors.

For those unfamiliar, mechanical thrombectomy involves physically removing a blood clot from a blocked artery in the brain using specialised devices delivered via a catheter. This procedure has revolutionized severe stroke care since its proven efficacy a decade ago. The SuperNOVA stent is a next-generation device designed to potentially offer improved performance in retrieving those tricky, large vessel occlusions, aiming for faster and more complete restoration of blood flow to the brain. The recent studies have confirmed its efficacy, showing high rates of successful recanalisation with excellent safety profiles for patients involved in the trials.

This breakthrough means clinicians may soon have another highly effective tool in their arsenal to combat strokes rapidly. Faster, more complete clot removal directly translates to better outcomes, reduced long-term disability and improved quality of life for survivors. Regarding its introduction for clinical practice in the UK, the path forward involves regulatory approval and NHS adoption processes. Following successful clinical trials, the device must receive the appropriate regulatory certification (such as a UKCA mark, previously CE mark, depending on current regulations) confirming it meets safety and performance standards.

Once certified, NHS trusts and National Institute for Health and Care Excellence (NICE) will need to evaluate the cost-effectiveness and clinical benefits compared to existing stent retrievers. This process can take time, often several months to a couple of years depending on the complexity and priority. So, while a promising development, ARNI Stroke Rehab UK comments that immediate availability in every UK hospital this spring is unlikely! Clinicians and patients can expect to see it introduced into specialist neurovascular centres gradually over the next one to three years, subject to successful regulatory hurdles and local commissioning decisions.

Imagine a future where stroke survivors can relearn brand-new signals and regain function using advanced technology that talks directly to their brain. Scientists have just achieved an incredible breakthrough: they’ve created a revolutionary, soft, wireless implant that uses tiny, gentle flashes of light to send information straight into the brain. The approach moves around the body’s traditional sensory pathways and instead interacts with neurons directly.

Published 4 days ago in Nature Neuroscience, the system is soft and flexible and fits beneath the scalp while resting on the skull. From this position, it can project carefully programmed light patterns through the bone to stimulate neurons across large areas of the cortex. During testing, scientists used tiny bursts of patterned light to activate specific groups of neurons in mouse models (these neurons are genetically modified to respond to light). The mice quickly learned that certain light patterns represented meaningful cues and used them to guide behaviour. Even though no normal senses were involved, the animals used these artificial signals to make decisions and complete behavioral challenges.

The fact that the implant is soft and wireless is also a key technical advantage; it reduces the likelihood of tissue damage and makes it much more practical for long-term use compared to rigid, wired alternatives. While this is still a cutting-edge research finding and not yet available for clinical use, researchers see broad potential for this approach. It could eventually support prosthetic limbs by supplying sensory feedback, deliver new types of artificial input for future hearing or vision devices, help manage pain without drugs, enhance recovery after injury or stroke, and even support brain-controlled robotic limbs.

A significant change is underway in how urgent stroke care will be delivered across Somerset, a development that impacts patients and healthcare professionals across the region. NHS Somerset Integrated Care Board has confirmed the closure and removal of the specialist hyper-acute stroke unit (HASU) at Yeovil District Hospital, a process scheduled to begin this spring. This strategic reconfiguration means that Yeovil will no longer receive patients experiencing an acute, emergency stroke event that requires immediate, hyper-acute treatment.

For residents in South Somerset, this change fundamentally alters the critical first steps of the stroke pathway. Instead of being admitted to Yeovil, patients identified as having an acute stroke will now be transported directly by ambulance to one of the larger, consolidated specialist units… the primary receiving centres will be Dorset County Hospital in Dorchester and Musgrove Park Hospital in Taunton.

This decision aligns with national NHS guidelines which strongly advocate for centralising hyper-acute stroke services into fewer, high-volume ‘centres of excellence’. The clinical rationale is robust: these larger units can maintain 24/7 access to highly specialised multidisciplinary teams, cutting-edge diagnostic equipment, and consistent thrombectomy services. Extensive national research demonstrates that patients treated in these dedicated HASUs have demonstrably better outcomes, including improved survival rates and reduced long-term disability, even if the ambulance journey is slightly longer. While the clinical evidence supports consolidation, the closure does raise valid concerns within the local community regarding increased travel times for families during a time of crisis..

It’s a chilling reminder that even those who appear robustly healthy can be silently walking a tightrope with their cardiovascular system. A case study published today in BMJ Case Reports brings to light the stark reality of the dangers associated with excessive energy drink consumption, even in an otherwise fit individual.

The subject was an active, healthy man in his 50s who maintained an active lifestyle, including running, and did not smoke or use recreational drugs. His single vice was a formidable one: a daily habit of consuming a staggering eight cans of energy drinks. This regimen pushed his daily caffeine intake to well over 1,200mg, significantly surpassing the recommended daily maximum of 400mg for a healthy adult.

The consequences were catastrophic. He was admitted to the hospital having suffered a severe stroke. Upon arrival, doctors recorded his blood pressure at an astronomical 254/150 mmHg (!); obviously a life-threatening hypertensive crisis. The sheer volume of caffeine, a potent vasoconstrictor and stimulant, placed immense and unsustainable stress on his vascular system, culminating in a cerebral vascular event.

The most sobering aspect of this story is the long-term impact. Once the man ceased consuming the energy drinks, his blood pressure normalised within a week. Yet, eight years later, he still lives with the permanent neurological deficit of numbness and weakness on his left side.

This case serves as a critical warning. It highlights the urgent need for both public awareness regarding the cardiovascular risks of these high-caffeine beverages and for healthcare professionals to be more proactive in inquiring about energy drink habits when diagnosing unexplained high blood pressure. The ‘healthy’ veneer of an active individual offered no protection against the cumulative toxic effects of this habit.

Not a News Item today, but an important Technical Reminder: for our stroke survivors and their professional therapists and ARNI neuro-instructors, navigating the complexities of recovery, understanding the mechanics of symptoms is critical. One of the questions that these groups ask experts in stroke is exactly why spasticity is negatively affected by movement speed.

So, one of the most defining characteristics of spasticity after stroke is its reliance on velocity – in effect, the question isn’t just if there is resistance, but how quickly you move. This phenomenon is rooted entirely in the intricate neurophysiology of the spinal cord reflex arc and the disruption caused by an upper motor neuron (UMN) lesion.

In a healthy system, our muscle tone is finely tuned by a balance of excitatory and inhibitory signals. After a UMN lesion, perhaps from a stroke or spinal cord injury, this descending inhibitory control is diminished or lost. The spinal circuitry becomes disinhibited, hypersensitive to input. The key sensory players here are the muscle spindles; specialised receptors within the muscle belly that detect changes in length and the speed of those changes.

The primary afferent fibers (Group Ia afferents) innervating these spindles are exquisitely sensitive to the velocity of a stretch. When a therapist or trainer moves a limb which is in in a position of spasticity, stretching the muscle, these velocity-sensitive fibers fire robustly. The faster the movement, the greater the electrical signal transmitted back to the spinal cord. In the disinhibited state, this heightened afferent barrage leads to an exaggerated excitation of the alpha motor neurons, the final common pathway for muscle contraction.

The result is an abnormally strong, brisk, exaggerated involuntary efferent contraction, which manifests clinically as increased resistance proportional to the speed of the stretch applied. This mechanism distinguishes spasticity from other forms of hypertonia like rigidity, which is velocity-independent. Understanding this principle is crucial for effective therapy; it’s why slow, sustained stretches are often more effective than quick movements in managing spasticity and why careful grading of speed is vital during functional tasks…

In the high-stakes world of stroke medicine, the mantra is simple and urgent: ‘Time is brain’. Every minute that a major blood clot blocks the flow to the brain, more vital tissue dies. Doctors race against the clock to diagnose, locate the blockage, and intervene. But what if we could give doctors an extra hour in that critical race? A groundbreaking new study suggests we can do exactly that, all thanks to an innovative artificial intelligence imaging tool.

Researchers have found that this sophisticated AI can help doctors spot deadly large vessel occlusions (LVOs);the most severe and debilitating type of stroke-causing clots, an average of more than an hour earlier than current clinical workflows allow.

The AI doesn’t replace the expert eye of a radiologist or neurologist; instead, it acts as an incredibly efficient triage system and second set of eyes. It rapidly processes complex brain scans and immediately flags critical findings, pushing relevant cases to the top of the queue for human review. This efficiency drastically cuts down the time from when a patient arrives at the emergency department to when the critical treatment decision is made…

Earlier diagnosis means faster initiation of life-saving treatments like mechanical thrombectomy (physically removing the clot). The difference between intervention at two hours versus three hours can mean the difference between a patient making a full functional recovery or suffering a severe, lifelong disability.

In our fast-paced world, stress is often dismissed as just ‘part of life’. We talk about it in passing, joke about our caffeine intake, and rarely view it as a serious health threat. However, emerging research is strengthening the vital connection between chronic, psychosocial stress and a significantly increased risk of stroke. This invisible emotional burden may be quietly damaging  brain.

Multiple extensive studies and large-scale meta-analyses published last month (Frontiers Neurol. 2025 Nov 6;16:1669925) , encompassing nearly a million (950,000) participants, confirm a moderate yet significant association between self-perceived chronic stress and stroke risk. The data is compelling, suggesting that stress impacts our health with a magnitude similar to factors like diabetes, making it a critical, and often overlooked, modifiable risk factor.

So, how does the pressure of a job or relationship translate into a physical blockage or bleed in the brain? The connection lies in the body’s ‘fight-or-flight’ response. When stressed, our systems flood with hormones like cortisol and adrenaline. While useful for escaping immediate danger, constant elevation of these hormones over years leads to sustained high blood pressure, chronic inflammation throughout the cardiovascular system, and actual damage to artery walls. This creates a perfect environment for clots to form and blood vessels to harden (atherosclerosis).

Furthermore, chronic stress often pushes people toward unhealthy coping mechanisms. Poor sleep quality, emotional eating, increased smoking, lack of physical exercise and higher alcohol consumption become common responses. These lifestyle choices independently pile on further risk factors for stroke, creating a vicious cycle of physical and emotional strain.

Intriguingly, recent research published in the journal Neurology® points to a potential gender disparity. One specific study focusing on young adults indicated that women reporting moderate to high stress levels faced a notably higher risk of stroke compared to men with similar stress burdens. This highlights a need for targeted awareness and stress management strategies tailored to different demographics.

The main takeaway is clear: managing stress is not just about mental well-being or ‘feeling better’… it’s a fundamental component of preventative medicine and physical health. Incorporating simple, consistent practices like mindfulness, regular walks, finding time for hobbies or seeking professional support can make a tangible, life-saving difference.

Frontiers Neurol. 2025 Nov 6;16:1669925 https://pmc.ncbi.nlm.nih.gov/articles/PMC12631615/

One of the biggest challenges for survivors is consistently performing the intensive physical therapy needed to regain movement.. especially for those dealing with hemiparesis, simple upper-limb exercises can feel impossible or deeply discouraging. Enter a brilliant innovation that’s changing the game: the Powered Rehab Skateboard, developed by Prof. Kenneth Fong and team at Hong Kong Polytechnic University.

The device is a sophisticated, motorised platform engineered specifically for the more-affected upper limb: it gently supports the arm and guides it through precise, repetitive therapeutic motions.

The core of the device is a wheeled, end-effector-based robotic system (a ‘skateboard’ on a table) which supports the patient’s arm and guides it through specific movement patterns (e.g., left-right, forward-backward, circular, and figure-eight motions)

It features an integrated torque sensor that detects the user’s active force in real-time. This allows the device to measure how much effort the patient is putting into the exercise. It also  uses a control system that interprets the sensor data to adjust the level of assistance dynamically. This system offers multiple operational modes: passive, assistive and resistive.

A machine learning algorithm (in advanced versions of the related research) captures activity data and adapts the therapy in real-time, ensuring optimal challenge and support for the individual’s specific stage of recovery. For home use, the device includes a micro edge detection sensor that triggers an alarm and stops operation if it approaches the edge of a table, preventing accidents.

In essence, the technology provides a ‘smart’ physical therapist at home, ensuring the patient receives the correct intensity and type of repetitive exercise necessary for neurorehabilitation and motor learning. 

It’s primarily an academic and clinical research innovation at this stage, so not yet a commercial product you can simply buy from a medical supply store today. To find out when it might become commercially available for UK stroke survivors, the best course of action is to follow updates here at ARNI – we’ll find out and let you know when we hear official announcements from Prof. Kenneth Fong’s research team. While we can’t buy it off the shelf yet, the promise it holds for home-based recovery is undeniable.